Clinical Chemistry Controls
Revvity Health Sciences Inc Cellaca PLX, anti-human CD8 APC Viability Kit, 100 Tests
Cellaca PLX, anti-human CD8 APC Viability Kit, 100 Tests
ABclonal Technology LDL Receptor (LDLR) Rabbit pAb
The low density lipoprotein receptor (LDLR) gene family consists of cell surface proteins involved in receptor-mediated endocytosis of specific ligands. The encoded protein is normally bound at the cell membrane, where it binds low density lipoprotein/cholesterol and is taken into the cell. Lysosomes release the cholesterol, which is made available for repression of microsomal enzyme 3-hydroxy-3-methylglutaryl coenzyme A (HMG CoA) reductase, the rate-limiting step in cholesterol synthesis. At the same time, a reciprocal stimulation of cholesterol ester synthesis takes place. Mutations in this gene cause the autosomal dominant disorder, familial hypercholesterolemia. Alternate splicing results in multiple transcript variants.
Genscript Corporation SAA MLB2 mAb Mouse
Serum Amyloid A (SAA) is an acute-phase protein The concentration of SAA in blood increases rapidly when tissue damage or inflammation occurs It is considered as an early biomarker for the diagnosis of many inflammatory diseases SAA can also be used as an important indicator of infection
Biolegend Purified anti-Apo E 109-116
Purified anti-Apo E, 109-116 [A17067B]; Isotype: Mouse IgG2b, κ; Reactivity: Human; Apps: WB, Direct ELISA; Size: 25 μg
Cayman Chemical 7-dehydro Cholesterol 50g
An immediate precursor to cholesterol; accumulates in Smith-Lemli-Opitz syndrome (SLOS); highly susceptible to free radical oxidation, giving rise to several oxysterols that may be involved in the pathogenesis of SLOS; levels are increased in brain, liver, and serum in a rat model of SLOS induced by the DHCR7 inhibitor AY 9944; converted to vitamin D3 by UVB light ex vivo
Cayman Chemical 7-dehydro Cholesterol 5g
An immediate precursor to cholesterol; accumulates in Smith-Lemli-Opitz syndrome (SLOS); highly susceptible to free radical oxidation, giving rise to several oxysterols that may be involved in the pathogenesis of SLOS; levels are increased in brain, liver, and serum in a rat model of SLOS induced by the DHCR7 inhibitor AY 9944; converted to vitamin D3 by UVB light ex vivo